Publications and Presentations

  1. Bhattacharjee S, Bose P, Patel K, Roy SG, Gain C, Gowda H, Robertson ES, Saha A*. (2018). Transcriptional and epigenetic modulation of autophagy promotes EBV oncoprotein EBNA3C induced B-cell survival. Cell Death Dis. 2018 May 22;9(6):605. * Corresponding Author.
  2. Ghosh Roy S, Robertson ES, Saha A*. (2016). Epigenetic impact on EBV associated B-cell lymphomagenesis. Biomolecules, 6(4), pii: E46. * Co-corresponding Author.
  3. Pei Y, Banerjee S, Sun Z, Jha HC, Saha A, Robertson ES. (2016). EBV nuclear antigen 3C mediates regulation of E2F6 to inhibit E2F1 transcription and promote cell proliferation. Plos Pathogens 12(8):e1005844. 
  4. Bhattacharjee S, Ghos Roy S, Bose P, Saha A*. (2016). Role of EBNA3-family proteins in EBV-associated B-cell lymphomagenesis. Front Microbiol, 7:457. * Corresponding Author.
  5. Saha A, Jha HC, Upadhyay SK, Robertson ES. (2015). Epigenetic silencing of tumor suppressor genes during in vitro Epstein-Barr virus infection. Proc Natl Acad Sci U S A, 112(37):E5199-207. 
  6. Dzeng RK, Jha HC, Lu J, Saha A, Banerjee S, Robertson ES. (2015). Small molecule growth inhibitors of human oncogenic gammaherpesvirus infected B-cells. Mol Oncol.; 9(2):365-76. 
  7. Jha HC, Aj MP, Saha A, Banerjee S, Lu J, Robertson ES. (2014). EBV essential antigen EBNA3C attenuates H2AX expression. J Virol.; 88(7):3776-88. 
  8. Saha A, Robertson ES. (2013). Insights into the Epstein-Barr virus nuclear antigen 3C mediated deregulation of cell-proliferation and apoptosis. Future Microbiol, 8(3):323-52. 
  9. Jha HC, Lu J, Saha A, Cai Q, Banerjee S, Prasad MA, Robertson ES. (2013). EBNA3C-mediated regulation of aurora kinase B contributes to Epstein-Barr virus-induced B-cell proliferation through modulation of the activities of the retinoblastoma protein and apoptotic caspases. J Virol.; 87(22):12121-38. 
  10. Banerjee S, Lu J, Cai Q, Saha A, Jha HC, Dzeng RK, and Robertson ES. (2013). The EBV latent antigen 3C inhibits apoptosis through targeted regulation of interferon regulatory factors 4 and 8. Plos Pathogens, 9(5):e1003314. 
  11. Jha HC, Upadhyay SK, Aj MP, Lu J, Cai Q, Saha A, Robertson ES. (2013). H2AX Phosphorylation Is Important for LANA-Mediated Kaposi's Sarcoma-Associated Herpesvirus Episome Persistence. J Virol, 87(9):5255-69. 
  12. Saha A, Lu J, Morizur L, Upadhayay SK, AJ Prasad M, Robertson ES. (2012). Epstein-Barr virus Nuclear Antigen 3C Blocks E2F1 Induced Apoptotic Pathway in EBV Infected Cells. Plos Pathogens; 8(3): e1002573. 
  13. Lu J, Verma SC, Cai Q, Saha A, Dzeng R, Robertson ES. (2012). Enhanced KSHV latent infection and proliferative capacity during primary infection. Plos Pathogens; 8(1):e1002479. 
  14. Cai Q, Guo Y, Xiao B, Banerjee S, Saha A, Lu J, Glisovic T, Robertson ES. (2011). Epstein-Barr virus Nuclear Antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis. Plos Pathogens; 7(12): e1002418. 
  15. Saha A, Halder S, Upadhayay SK, Lu J, Kumar P, Murakami M, Cai Q, Robertson ES. (2011). EBNA3C Facilitates G1-S Transition by Stabilizing and Enhancing the Function of Cyclin D1. Plos Pathogens; 7(2):e1001275. 
  16. Saha A, Bamidele A, Murakami M, Robertson ES. (2011). EBNA3C Attenuates the Function of p53 through Interaction with the Inhibitor of Growth Family Proteins, 4 and 5. J Virol.; 85(5):2079-88.  
  17. Saha A, Robertson ES. (2011). Epstein-Barr virus in B-cell Lymphoma: Pathogenesis and Clinical Outcomes. Clin Cancer Res.; 17(10):3056-63. 
  18. Saha A, Robertson ES. (2011). Functional Modulation of the Metastatic Suppressor Nm23-H1 by Oncogenic Viruses. FEBS Lett.; 585(20):3174-84. 
  19. Saha A*, Kaul R*, Murakami M, Robertson ES. (2010). Tumor viruses and cancer biology: modulating signaling pathways for therapeutic intervention. Cancer Biol Ther.; 10(10):961-78.*Equal Contribution. 
  20. Xiao B, Verma SC, Cai Q, Kaul R, Lu J, Saha A, Robertson ES. (2010). Bub1 and CENP-F can contribute to Kaposi's sarcoma-associated herpesvirus genome persistence by targeting LANA to kinetochores. J Virol.; 84(19):9718-32. 
  21. Kumar P*, Saha A*, Robertson ES. (2010). Epstein-Barr virus Hijacks Cell-Cycle Machinery: EBV can perturb cellular pathways, contributing to the development of cancer. Microbe; 5: 1-6. *Equal Contribution. 
  22. Yi F*, Saha A*, Murakami M*, Kumar P, Knight JS, Cai Q, Choudhuri T, Robertson ES. (2009). Epstein-Barr virus nuclear antigen 3C targets p53 and modulates its transcriptional and apoptotic activities. Virology; 388(2):236-47. *Equal Contribution. 
  23. Saha A, Murakami M, Kumar P, Bajaj B, Sims K, Robertson ES. (2009). Epstein-Barr virus nuclear antigen 3C augments Mdm2-mediated p53 ubiquitination and degradation by deubiquitinating Mdm2. J Virol.; 83(9):4652-69. 
  24. Kumar P, Murakami M, Kaul R, Saha A, Cai Q and Robertson ES. (2009). Deregulation of Cell Cycle Machinery by the Epstein-Barr virus Nuclear Antigen 3C. Future Virol.; 4(1):1-8. 
  25. Mitra G, Saha A, Gupta TD, Poddar A, Das KP, Das Gupta SK, Bhattacharyya B. (2007). Chaperone-Mediated Inhibition of Tubulin Self-Assembly. Proteins; 67(1):112-20. 
  26. Sharma A, Saha A, Bhattacharjee S, Majumdar S, Das Gupta SK. (2006). Specific and Randomly Derived Immunoactive Peptide Mimotopes of Mycobacterial Antigens. Clin Vaccine Immunol.; 13(10):1143-54. 
  27. Saha A, Sharma A, Dhar A, Bhattacharyya B, Roy S, Das Gupta SK. (2005). Antagonists of Hsp16.3, A Low-Molecular-Weight Mycobacterial Chaperone and Virulence Factor, Derived from Phage-Displayed Peptide Libraries. Appl Environ Microbiol.; 71(11):7334-44.